In the past, people have contracted hepatitis C through blood transfusions.As of 2003, cirrhosis resulting from chronic hepatitis has emerged as a leading cause of death among HIV-positive patients; in Europe, about 30% of HIV-positive patients are coinfected with a hepatitis virus.These symptoms are related to ammonia intoxication and the failure of the liver to convert ammonia to urea.
The whites of the eyes and the skin may turn yellow (jaundice), and urine may be dark yellow or brown. Sometimes the patient develops persistent high blood pressure due to the scarring (portal hypertension). It can cause veins to enlarge in the stomach and in the tube leading from the mouth to the stomach (esophagus).
These enlarged veins are called varices, and they can rupture and bleed massively.
If the liver loses its ability to remove toxins from the brain, the patient may have additional symptoms.
The patient may become forgetful and unresponsive, neglect personal care, have trouble concentrating, and acquire new sleeping habits.
Liver injury, reactions to prescription medications, exposure to toxic substances, and repeated episodes of heart failure with liver congestion can cause cirrhosis.
The disorder can also be a result of diseases that run in families (inherited diseases) like: Obesity has recently been recognized as a risk factor in nonalcoholic hepatitis and cirrhosis.
Their disease is detected during a routine physical or when tests for an unrelated medical problem are performed.
This type of cirrhosis can also be detected when complications occur (decompensated cirrhosis).
Cirrhosis changes the structure of the liver and the blood vessels that nourish it.